Belgium To Approve Euthanasia of Alzheimer Patients and Autistic Children

A proposed law on the verge of approval by the Belgium parliament would allow children to decide for themselves whether they should be euthanized ("killed") by medical personnel. Currently, Belgian law limits euthanasia to persons 18 and older, but with the rise of autistic children thanks to biopesticides, GMOs and vaccines, nations are increasingly trying to figure out what to do with all these children who have been permanently damaged by the medical and biotech industries.

The answer, of course, is to simply kill them. It's difficult to kill children off under current law without being charged with murder, however. So this proposed new law would allow doctors to decide whether children of any age (yes, even a five-year-old) can, themselves, "consent" to being euthanized without parental consent.

The mercy killing of autistic children has already begun in the UK, by the way. As the Daily Mail reports: "Desperate mother and godmother 'killed severely autistic boy, 14, by stabbing him multiple times in the chest' when they became overwhelmed caring for him 24/7 after demanding he be removed from the hospital and in put in their care."

Not surprisingly, this "mercy killing" bill was introduced by the socialist party, since socialists tend to hate humanity no matter what country they live in. The proposed legislation calls for, "the law to be extended to minors if they are capable of discernment or affected by an incurable illness or suffering that we cannot alleviate," reports AFP.

That story goes on to report, "Socialist Senator Philippe Mahoux, who helped draft the proposed changes, said there had been cases of adolescents who 'had the capacity to decide' their future."

Because, of course, a six-year-old isn't old enough to drive a car or buy a beer, but they can certainly consent to being killed by the state... especially if they're already a burden on health care costs.

As the law proponents claim, it "marks a turning point in the nation's approach to the rights of young people, some of whom would be able to choose to die if the law were to pass, even while still being legally barred from driving, marrying, voting or drinking liquor until they turned 18," reports IBtimes.com

(Story continues below...)

Alzheimer's patients to also face euthanization under proposed law

But wait, there's more! It's not just children who will be euthanized under this new law, it's also scores of Alzheimer's patients.

"Parliamentarians would also consider extended mercy-killing to people suffering from Alzheimer's-type illnesses," said the socialist senator.

Euthanasia is, of course, the most convenient way for any government to get rid of people it doesn't want to care for. Instead of teaching the public how to prevent and reverse Alzheimer's disease, the government simply passes a new law to murder them all while calling it "compassionate medicine."

The age of mass murder by government is upon us. It won't be long before such laws spread to other nations and are expanded to people with "mental health problems" or even cancer. While in theory the idea of euthanasia may have merit in a strictly limited scope, the truth is that governments are now jumping on the euthanasia bandwagon to provide attempted legal justification for the mass murder of children and senior citizens whom they no longer wish to support with health care expenditures. Save the government from bankruptcy! Have grandma euthanized! (Right alongside little autistic Johnny, too...)

Governments, after all, are ultimately political machines of death and destruction, which explains why all the wars fought in the 20th century were started by governments, not the People. War is simply another way to accomplish the mass killing that the twisted, demented people at the top of every government truly enjoy.

And there's nothing more delicious to these power freaks than killing little children. If you can take a five-year-old into a doctor's office and pump his veins full of deadly chemicals while calling it a "mercy killing," it's almost erotic to these power-hungry control freaks. Too bad the BBC's Jimmy Savile isn't still around, or he might even find something else to do with the children's corpses afterward.

Sick? You bet it is. We're talking about the state-sponsored murder of children here. There's nothing polite about it. You can also bet this will become the new way that governments dispose of autistic children and then claim rates of autism are "going down" because not as many children have it anymore. (Yeah, after they've all been euthanized.)

Governments, you see, will even resort to the mass murder of children to protect the vaccine industry. And what is the vaccine industry other than a child-killing monstrosity to begin with? That's why the industry loves to run dangerous vaccine experiments on little children, too. There's nothing quite as rewarding to these vaccine pushers as seeing a high-IQ child turned into a drooling, screaming, zombified autism case in the hours after receiving a vaccine injection.

God help us all. We are all being mass murdered by the fascist global state, and most of the population is too dumbed-down to realize what's happening.

Does Eating Junk Food Also Contribute To Dementia?

Alzheimer’s is one of the major medical problems of our age. 

 

A ghastly disease that condemns millions to years of confusion and fear, it also threatens to bankrupt our health system — yet doctors don’t really know what causes it, or how to treat it effectively.

 

But could that be about to change?

Scientists increasingly believe Alzheimer’s is linked to type 2 diabetes — so closely linked, in fact, it’s even being called ‘brain diabetes’ or type 3 diabetes.

 

This surprising new theory holds out hope that treatments already available for diabetes may also be able to help dementia sufferers, slowing down or stopping the progression of the disease.

 

When it comes to type 2 diabetes, there’s no mystery about what’s behind the soaring rates — eating too much, especially junk food that’s packed with sugar, refined carbohydrates and fat.

This leads to damagingly high levels of sugar in the blood and high levels of insulin needed to clear it away. The rising levels of glucose and insulin affect the blood vessels of the heart and extremities, potentially leading to blindness and amputations.

 

But it seems raised insulin levels also affect the brain.

 

Recent research has found this hormone plays a much more important role in the brain than once thought — protecting cells and helping to lay down memories.

 

The key to diabetes, and very possibly to Alzheimer’s, is insulin resistance. 

 

This is when your body becomes so used to extra insulin in the bloodstream that it needs more and more to have the same effect. 

If you constantly eat high glycaemic foods, such as cakes and white bread, this pushes up the amount of sugar in your blood. Insulin has to work overtime, turning glucose into fat and sweeping it into storage.

The favourite storage areas — fat cells, muscles and the liver — start to reject insulin’s fat deliveries.Gradually, things start to go wrong. Your body has to keep making more insulin to make the normal store areas respond. 

 

This is turning out to be especially damaging in the brain.

 

Insulin is so important to the proper function of the brain that it makes its own supplies.

The hormone keeps the brain’s blood vessels healthy and also helps brain cells (neurons) absorb the sugar they need to function. 

 

This allows them to change in response to learning and to lay down memories. 

The hormone is also involved in making some of the chemicals that pass messages around the brain.

 

But if glucose levels keep shooting up after meals, insulin resistance kicks in, and the relationship between neurons and insulin starts to break down — and the brain suffers damage.

For instance, insulin resistance is being linked with formation of the plaques — deposits of damaged protein —  that are a classic sign of Alzheimer’s. 

 

In a study at Brown University in the U.S., when insulin supply in rats’ brains was blocked — mimicking the effects of insulin resistance — the animals became disoriented and plaques appeared in their brain cells. 

 

The effects of raised sugar are not limited to rats. 

 

Last year, a small study from the U.S. Department of Veteran Affairs looked at healthy volunteers who had been on a junk-food diet for only four weeks. They had a raised level of the markers for plaque in their spinal fluid. 

 

Scientists are still finding out what happens when insulin levels in the brain rise. 

‘But we know it is much more important than we thought,’ says Professor Jennie Brand-Miller, a biochemist at the University of Sydney and world authority on insulin (she helped develop the glycaemic index).

 

High levels of insulin could also be having a damaging effect on neurons, she says. 

‘This is because insulin comes partnered with another hormone, amylin, which makes the same sort of plaques as those found in the brains of dementia patients, except in the pancreas.

'It could be contributing to plaque formation in the brain.’

 

High sugar levels don’t just push up insulin — they can also damage the brain directly. 

 

Last May, researchers at the University of California showed for the first time that eating high levels of fructose, the concentrated sweetener found in many processed foods, reduced brain function in rats. 

 

‘A high fructose diet over the long term alters your ability to learn and remember information,’ says research leader, Fernandez Gomez-Pinilla, a professor of neurosurgery.

Earlier this year, an Australian team scanned the brains of 300 older people who didn’t have dementia.

 

The brains of those with diabetes shrank up to two-and-a-half times faster than normal. 

This shrinking occurred most in the frontal lobe that controls many functions damaged by Alzheimer’s — decision-making, emotional control and long-term memory.

 

What makes the theory linking type 2 diabetes and Alzheimer’s so convincing is that both are increasing at similar rates.

 

In other words, it’s very likely the same process is behind the two. 

 

Dr Suzanne de la Monte, a neuropathologist at Brown University, says: ‘Before 1980, there was little overlap between Alzheimer’s and diabetes. 'In fact, until then diabetes rates had been declining.’ Then something changed.

 

‘In every age group, the death rate from both diseases in 2005 is much higher than in 1980. 

'I believe Alzheimer’s starts with insulin resistance,’ says Dr de la Monte. 

If, indeed, the links between diabetes, insulin and Alzheimer’s stand up, the good news is that it will give us more treatment options.

 

Drugs already used to treat diabetes might benefit Alzheimer’s patients or even stop the disease developing.As it happens, it’s not the usual diabetes drugs — such as metformin — that may help.

 

‘So far, giving the older insulin drugs to Alzheimer’s patients hasn’t proved effective,’ says Dr Callum Sutherland, a specialist in insulin action at Dundee University. 

The latest approach, in fact, is to boost insulin levels in the brain directly. 

 

A small-scale trial last year found that dementia patients’ memories improved after nasal sprays of insulin. 

 

A larger trial on 240 patients with early signs of dementia is under way at Washington University, in Seattle, to see if it can slow the disease down.

 

Research with a new type of diabetes drug, known as GLP-1, is promising. 

It slows down brain damage in mice with dementia.

 

Of course, the insulin theory also gives us even more incentive to eat a diet that keeps refined carbohydrates to a minimum.

 

There is still work to be done to understand all the  connections, as not everyone with diabetes develops Alzheimer’s and vice versa, says Dr Sutherland.

 

But finding out exactly how this link works could mark a turning point in the fight against this dreadful disease.

Surprising Ways To Tell If You Might Develop Alzheimers

The statistics on Alzheimer’s disease are overwhelming. More than 5 million Americans are living with Alzheimer’s disease. One in eight older Americans has the condition, and Alzheimer’s is the sixth leading cause of death in the United States.



But there is another little-known stat that is equally, if not more, startling. Among cognitively normal people over the age of 75, about one-third has sufficient amyloid plaques and neurofibrillary tangles to meet the criteria for Alzheimer’s disease.

These plaques and tangles are especially prominent in those who have Alzheimer’s and contribute to the degradation of neurons in the brain. But they also develop in many elderly people, even those who do not have Alzheimer’s.

It’s these cases — referred to as asymptomatic Alzheimer’s disease (ASYMAD) — that researchers find particularly fascinating. People with ASYMAD have all the physical characteristics that could lead to the progression of Alzheimer’s disease, but have managed to avoid it. If researchers figure out how, then that could lead the way to better, more effective prevention and treatment measures for Alzheimer’s, which are both sorely lacking right now.

Negativity Hurts the Brain

A team of researchers recently hypothesized that certain personality traits contribute to the cognitive resilience of ASYMAD. They compared the personality traits of those who subsequently developed clinical dementia with those who subsequently died cognitively normal, but with plaques and tangles indicative of Alzheimer’s discovered at autopsy.

And the results of this study may just want to make you turn that frown upside down, especially if you want to protect your brain.

The subjects in this study were also participants in the Baltimore Longitudinal Study of Aging, a study of physical and psychological aging. All agreed to have an autopsy of the brain following their death.

Researchers analyzed data obtained from 111 of the participants who completed personality tests at least once before the onset of cognitive decline, and who underwent autopsies after
death.

Personality traits were measured using a 240-item questionnaire that looked at 30 facets of personality, six for each of the five major dimensions of personality: neuroticism, extraversion, openness, conscientiousness and agreeableness.

As for neuropathology, participants were divided into three groups based on their cognitive health in their last year of life and their autopsy results:

1. Normal: Patients had no history of cognitive decline and autopsies showed no amyloid beta plaques or other signs of Alzheimer’s.

2. ASYMAD: Patients had no history of cognitive decline, but the autopsies showed amyloid beta plaques in the brain.

3. AD: Patients received clinical diagnosis of dementia and/or Alzheimer’s disease while alive, and autopsies revealed amyloid beta plaques and other signs indicative of Alzheimer’s.
Looking at the results of the autopsies and the personality questionnaires, researchers discovered that those who had greater emotional resilience and conscientiousness had lower risk of developing dementia, even if their autopsies revealed plaques or other signs of Alzheimer’s.

More specifically, ASYMAD patients scored lower on their personality questionnaires on negative traits like neuroticism, vulnerability to stress, anxiety and depression, compared with controls and to those who actually developed Alzheimer’s disease.
In addition, low scores on conscientiousness were strongly associated with the development of clinical dementia.

Researchers believe those who are more emotionally stable and conscientious may have greater resilience because they are generally healthier and engage in behaviors that reduce the risk of dementia — like exercising and abstaining from tobacco use. In addition, emotionally stable people tend to have better metabolic and inflammatory risk profiles and are less likely to have depression — all of which have been linked to dementia.

Also, they found that personality might be related to the severity of disease. Specifically, high neuroticism and low agreeableness were linked to more advanced spread of tangles. And skepticism, cynicism and being manipulative/deceptive were the characteristics associated with the most severe amyloid plaques and tangles.

This new knowledge of how personality traits affect the development of dementia allows us to see just how much of a role attitude plays in our overall health.

The more negative your overall demeanor, the more it will affect your health, possibly leading to cognitive decline and Alzheimer’s disease. Conversely, even if you are at physically high risk of Alzheimer’s, having a positive attitude toward life could delay or stop the progression of the disease. All the more reason to spend as much time as you can smiling, laughing and appreciating all the little things in your life.

Sources:
Terracciano A et al. Personality and resilience to Alzheimer’s disease neuropathology: a prospective autopsy study. Neurobiol Aging. 2012 Oct 2. pii: S0197-4580(12)00430-7. doi: 10.1016/j.neurobiolaging.2012.08.008. [Epub ahead of print.]
Saczynski JS et al. Depressive symptoms and the risk of dementia: the Framingham Heart Study. Neurology. 2010 Jul 6;75(1):35-4

2 Billion Dementia Patients?

2 BILLION Dementia Patients? Medical Establishment Shocked by At-Risk Numbers on the "Beyond comparison with anything that has been faced during the entire history of humanity," leading doctor says. October 11, 2012 | No one--to repeat, no one--understands how it happens. Every day, the minds of millions of high-functioning people slowly slip into another reality, one in which life events, loved ones' faces, children’s names and all the other memories that constitute a person’s identity have disappeared. Deep inside their brains, the slide begins decades before the symptoms of dementia manifest. By the time the car keys begin to go missing, it’s already far too late. Currently, there are about 36 million people in the world suffering from dementia. By 2050, the population at risk for the disease is expected to hit two billion, causing what experts say will be an unprecedented health care crisis that could cost $1 trillion a year in the U.S. alone. "The scope of the looming medical-care disaster is beyond comparison with anything that has been faced during the entire history of humanity,” said Dr. Barry Greenberg, the director of strategy for the Toronto Dementia Research Alliance, in a recent talk in Canada. Researchers have spent decades and tens of billions of dollars in searching for a treatment to Alzheimer’s, to no avail. Even before the looming health care crisis became clear, a drug that could slow or halt the chronic, terminal evaporation of a person’s memory has long been considered the Holy Grail of neuroscience research. The reasons are both economic and philosophic. Since the protracted disease is often as painful for relatives as it is for the patient, drugs are in high demand, with pharmaceutical companies projecting a $14 billion a year market by 2020. But the real reason that a treatment is so highly desired may be because the disease’s challenges the fundamental modern conception of humanity. If Descartes’ proposition, “I think, therefore I am” is accepted as what it means--at least in the Western world--to be human, then what about the projected two billion people who will soon become living, breathing entities without the capacity to remember their own thoughts? Today, many Alzheimer’s experts are blunt about the lack of any available treatments. "There really is nothing," Dr. Evelyn C. Granieri told Technology Review in its recent feature on the state of Alzheimer's research. "You don't get better, ever." This medical reality hasn’t stopped the pharmaceutical industry from turning a profit by selling drugs that are, at best, only minimally effective. Since 1996, Japanese company Eisai and U.S. drug marketer Pfizer have raked in more than $2 billion on the drug Aricept, even though studies have shown the medicine to barely slow the disease. In 2012, the Federal Drug Administration caused a scandal when it approved a higher dosage of the drug--called Aricept 23 mg--so that Pfizer and Eisai could extend their copyright despite evidence that the higher dosage was no more effective and was more likely to cause gastrointestinal problems for patients. Meanwhile, scientists and federal agencies are rushing to find a real treatment that could stave off the economic and social disaster of having a world population of two billion patients who require daily care. Last February, the National Institute of Health approved additional Alzheimer’s research funding. “We can’t wait to act,” Kathleen Sebelius, the U.S. health and human services secretary, said upon the announcement. A new round of clinical trials focused on preventing the onset of the disease--rather than treating it once it has already begun--are beginning this winter. Scientists hope that these trials will demonstrate that the disease’s progression can be interrupted, that the chronic deterioration of patients’ memories can be stopped if the disease is attacked early enough. This goal faces many challenges. There are numerous genetic mutations of Alzheimer’s, which requires scientists to match clinical trials with the appropriate patients. The disease often lies dormant for decades in a patient’s cerebral spinal fluids before it begins to affect one’s cognitive functioning, prompting scientists to ask just how early the intervention must occur. And all the while, the clock is ticking--not just for individual patients, but also for the entire health care system, prompting some scientists to call for a suspension of the private, profit-based research model in favor of a global, public-private program. "The medical-care system is going to be bankrupt by 2050 if we don't figure out a way to delay or treat Alzheimer's disease," Greenberg told Technology Review. "The competitive marketplace," he says, "was not conceived to overcome problems of this magnitude."